Preliminary reports suggest conolidine may inhibit specific ion channels, decreasing neuronal excitability and restricting discomfort indicators. This mechanism is especially pertinent in neuropathic pain, wherever abnormal signaling results in persistent pain. Moreover, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to agony perception. Computational d... https://rone824dto0.wikilima.com/user