Preliminary studies point out conolidine may inhibit unique ion channels, minimizing neuronal excitability and limiting soreness signals. This mechanism is especially related in neuropathic pain, in which abnormal signaling triggers persistent distress. Also, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to soreness perception. Computational docking re... https://samuelm011qdp8.wikipublicist.com/user